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primary antibodies against psa-ncam  (Cell Signaling Technology Inc)


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    Cell Signaling Technology Inc primary antibodies against psa-ncam
    Primary Antibodies Against Psa Ncam, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/primary+antibodies+against+psa-ncam/primary+antibodies+against+psa+ncam/pm30341743-87-26-30
    Average 90 stars, based on 1 article reviews
    primary antibodies against psa-ncam - by Bioz Stars, 2026-10
    90/100 stars

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    Related Articles

    Immunocytochemistry:

    Article Title: Deficiency of the clock gene Bmal1 affects neural progenitor cell migration.
    Article Snippet: For immunocytochemistry the cells were incubated with 10% normal goat serum in PBS-T 0.2% at RT for 1 h, followed by an incubation with the primary antibodies against PSA-NCAM (1:200, Cell Signalling Technologies, Danvers, MA, USA), GFAP (1:500, DAKO; Denmark), DCX (1:1000, Abchem, UK), or 8-OH(d)G (1:250, QED Bioscience, CA, USA) for 12 h at RT.

    Incubation:

    Article Title: Deficiency of the clock gene Bmal1 affects neural progenitor cell migration.
    Article Snippet: For immunocytochemistry the cells were incubated with 10% normal goat serum in PBS-T 0.2% at RT for 1 h, followed by an incubation with the primary antibodies against PSA-NCAM (1:200, Cell Signalling Technologies, Danvers, MA, USA), GFAP (1:500, DAKO; Denmark), DCX (1:1000, Abchem, UK), or 8-OH(d)G (1:250, QED Bioscience, CA, USA) for 12 h at RT.



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    Image Search Results


    Reduced neurogenesis marker proteins 72 h after status epilepticus in P10 rat pups. Graphs show representative immunoblots for neurogenesis marker proteins (A) PSA-NCAM, (B) NeuroD, (C) DCX and (D) TUC-4, in vehicle-injected controls (CON) and rat pups that underwent status epilepticus induced by intra-amygdala KA (SE). Tubulin is included as a guide to loading. Unpaired t test with Welch’s correction. *P < 0.05; **P < 0.01 (n = 3 per group). TUC-4 (non-significant; P = 0.88).

    Journal: International Journal of Physiology, Pathophysiology and Pharmacology

    Article Title: Neurogenic function in rats with unilateral hippocampal sclerosis that experienced early-life status epilepticus

    doi:

    Figure Lengend Snippet: Reduced neurogenesis marker proteins 72 h after status epilepticus in P10 rat pups. Graphs show representative immunoblots for neurogenesis marker proteins (A) PSA-NCAM, (B) NeuroD, (C) DCX and (D) TUC-4, in vehicle-injected controls (CON) and rat pups that underwent status epilepticus induced by intra-amygdala KA (SE). Tubulin is included as a guide to loading. Unpaired t test with Welch’s correction. *P < 0.05; **P < 0.01 (n = 3 per group). TUC-4 (non-significant; P = 0.88).

    Article Snippet: Sections at the level of dorsal hippocampus from control and KA rats at P55 were fixed, blocked and incubated with primary antibodies against PSA-NCAM (Monoclonal; Chemicon) followed by secondary antibodies coupled to AlexaFluor 568 (BioSciences) and mounted with 4’,6-diamidino-2-phenylindole (DAPI; Vector laboratories).

    Techniques: Marker, Western Blot, Injection

    Summary of immunoblot data on neurogenesis gene responses at 3 and 45 days after status epilepticus in P10 rats

    Journal: International Journal of Physiology, Pathophysiology and Pharmacology

    Article Title: Neurogenic function in rats with unilateral hippocampal sclerosis that experienced early-life status epilepticus

    doi:

    Figure Lengend Snippet: Summary of immunoblot data on neurogenesis gene responses at 3 and 45 days after status epilepticus in P10 rats

    Article Snippet: Sections at the level of dorsal hippocampus from control and KA rats at P55 were fixed, blocked and incubated with primary antibodies against PSA-NCAM (Monoclonal; Chemicon) followed by secondary antibodies coupled to AlexaFluor 568 (BioSciences) and mounted with 4’,6-diamidino-2-phenylindole (DAPI; Vector laboratories).

    Techniques: Western Blot

    Restitution of neurogenesis marker proteins at P55 in rats that underwent status epilepticus at P10. Graphs and representative immunoblots for a selection of neurogenesis marker proteins (A) PSA-NCAM, (B) NeuroD, (C) DCX and (D) TUC-4 in the hippocampus of P55 rats that received either vehicle (CON) or intra-amygdala KA to induced status epilepticus (SE) at P10. Data were analysed using unpaired t test. Tubulin is included as a guide to loading. Unpaired t test with Welch’s correction (n = 4 per group). PSA-NCAM (P = 0.34); NeuroD (not tested/no signal); DCX (P = 0.07); TUC-4 (P = 0.21). (E) Representative immunostaining for PSA-NCAM showing similar level of staining in a CON and a rat with hippocampal sclerosis at P55. h, hilus; sgz, subgranular zone; gcl, granule cell layer. Bar, 70 μm.

    Journal: International Journal of Physiology, Pathophysiology and Pharmacology

    Article Title: Neurogenic function in rats with unilateral hippocampal sclerosis that experienced early-life status epilepticus

    doi:

    Figure Lengend Snippet: Restitution of neurogenesis marker proteins at P55 in rats that underwent status epilepticus at P10. Graphs and representative immunoblots for a selection of neurogenesis marker proteins (A) PSA-NCAM, (B) NeuroD, (C) DCX and (D) TUC-4 in the hippocampus of P55 rats that received either vehicle (CON) or intra-amygdala KA to induced status epilepticus (SE) at P10. Data were analysed using unpaired t test. Tubulin is included as a guide to loading. Unpaired t test with Welch’s correction (n = 4 per group). PSA-NCAM (P = 0.34); NeuroD (not tested/no signal); DCX (P = 0.07); TUC-4 (P = 0.21). (E) Representative immunostaining for PSA-NCAM showing similar level of staining in a CON and a rat with hippocampal sclerosis at P55. h, hilus; sgz, subgranular zone; gcl, granule cell layer. Bar, 70 μm.

    Article Snippet: Sections at the level of dorsal hippocampus from control and KA rats at P55 were fixed, blocked and incubated with primary antibodies against PSA-NCAM (Monoclonal; Chemicon) followed by secondary antibodies coupled to AlexaFluor 568 (BioSciences) and mounted with 4’,6-diamidino-2-phenylindole (DAPI; Vector laboratories).

    Techniques: Marker, Western Blot, Selection, Immunostaining, Staining